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quote:Originally posted by hawaiigirl413 I was diagnosed with MS 10 years ago... auto immune problems with (lupus?)
quote:Originally posted by sdurkee34has anyone heard that the darker your skin the color the more myelin you have on your axons thus creating quicker reactions from the central nervous system, which allows better explosive movement such as jumping by people of color? My old coach who has his phd in excercise science said that one of his grad school professors had said this but i have been unable to find any written sources of this?steve durkee
quote:Originally posted by ROBERTAnyone researching therapies for MS should be aware that MS can follow a "relapsing-remitting" pattern. During remission there is a marked recovery, e.g. sufferers can regain the vision they have lost, and the ability to walk. This recovery is due to remyelination which occurs naturally.If a therapy was used during this remission period it would be possible to wrongly attribute the recovery to the therapy , when in fact it was naturally occuring, and the therapy useless or even harmful.
quote:Originally posted by BassAndrewI recently read (somewhere?) that there is a statistically higher incidence of MS for individuals that had mononucleosis in their youth. The paper did not suggest that the epstein-barr virus was to blame for the development of MS, but rather that it somehow altered the immune system. Have you seen any research on this- if so, do you have any comments?Several acquaintances of mine either have MS or have family members with MS- so I find this whole thread fascinating.
quote:Originally posted by ikoThe old cod liver oil could help restoring immune balance, but it would probably take some time (several months).I wasn't able to find clinical results about vitamin A,D and omega-3 (the main components of cod liver oil)in MS...but I'll go on surfing PubMed!
http://jnnp.bmj.com/cgi/rapidpdf/jnnp.2008.157164v1.pdfChronic cerebrospinal venous insufficiency in patients with multiple sclerosisPaolo Zamboni 1*, Roberto Galeotti 1, Erica Menegatti 1, Anna M Malagoni 1, Giovanna Tacconi 1, Sergio Dall'ara 1, Ilaria Bartolomei 2 and Fabrizio Salvi 21 Vascular Diseases Center, University of Ferrara, Italy2 Neurology, Bellaria Hospital, Bologna, ItalyAccepted 10 November 2008* AbstractBackground: The extracranial venous outflow routes in clinically defined multiple sclerosis (CDMS) have never been investigated.Methods: Sixty-five patients affected by CDMS, and 235 controls composed, respectively, of healthy subjects, healthy subjects older than CDMS patients, patients affected by other neurological diseases, and older controls not affected by neurological diseases but scheduled for venography (HAV-C), blindly underwent a combined transcranial and extracranial Color-Doppler high-resolution examination (TCCS-ECD) aimed at detecting at least two of five parameters of anomalous venous outflow. According to the TCCS-ECD screening, patients and HAV-C further underwent selective venography of the azygous and jugular venous system with venous pressure measurement.Results: CDMS and TCCS-ECD venous outflow anomalies were dramatically associated (OR 43, 95% CI 29-65, p<0.0001). Subsequently, venography demonstrated in CDMS, and not in controls, the presence of multiple severe extracranial stenosis, affecting the principal cerebrospinal venous segments; it configures a picture of chronic cerebrospinal venous insufficiency (CCSVI) with four different patterns of distribution of stenosis and substitute circle. Moreover, relapsing-remitting and secondary progressive courses were associated to CCSVI patterns significantly different from those of primary progressive (p<0.0001). Finally, the pressure gradient measured across the venous stenosies was slightly but significantly higher.Conclusion: CDMS is strongly associated with CCSVI, a picture never been described so far, characterized by abnormal venous haemodynamics determined by extracranial multiple venous strictures of unknown origin. The location of venous obstructions plays a key role in determining the clinical course of the disease.This paper is very exciting for myself and people with multiple sclerosis.http://answers.google.com/answers/threadview/id/271074.html here is another doctor who is convinced that MS is caused or at least contributed to by fluctuations in venous pressure. Franz Alfons Schelling, MD
Is there anything that IBT doesn't work for?