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WOW! Ikod,I am stunned. I like you have a connection to leukemia and children. Don't give up keep pushing someone will listen... I am listening. I want to talk to you about this very fascinating area on COD Liver Oil.I am a Pedi Oncologist in USA... we don't often talk about alternative medical practice/methods...but I agree with you. We cannot be satisfied with the stalemate of the last several years.Best wishes.
Speaking of why giving stinky "cod" instead of specific synthetic substances, let's borrow this note from the anti-oxidant topic of the Forum:quote: A quote from the article is "Just because a food with a certain compound in it is beneficial to health, it does not mean a pill with the same compound in is"That's exactly right. A pill sometimes works better than the original food and viceversa.Scientists versus Mother Nature and her tricksIn the late '70s researchers opened their enormous freezers where thousands of serum samples from blood donors had been stocked since over 10yrs before. They wanted to test vitamin A concentration (knowing that it is well preserved in frozen samples) and look for a correlation with cancer incidence in those individuals. Experimental data in animals had demonstrated a positive effect of retinoic acid on precancerous lesions.They found a strong inverse relation between vitamin A concentration and risk of tumor. All the media started recommending vitamin A to prevent or even fight cancer.Few years later a proper RCT (randomized clinical trial) was started: a group of nurses and doctors took either a certain dose of vitamin A or a placebo every day for years. The conclusion of the study was disappointing: no difference in cancer incidence with or without vitamin A.Some clever mind offered an explanation for this: vitamin A had been found increased in blood donors who had lower risk of cancer because it had been eaten together with some other more effective anticancer compounds.Here we go with all the broccoli, cabbage, cauliflowers and so on...they are rich of vitamin A and probably have other mysterious anticancer factors.ikoAddendum:Vitamin A instead of cod liver oil would play the same trick...if you gave vit.A to patients because the ones taking 'cod' had higher levels of retinoic acid in their blood and were doing better (hypothesis!), you could get poor results because you are not giving together Vit.D and a bit of omega-3 fatty acids, the original recipe.:mudneddA Vitamin D instead of cod liver oil would play the same trick...if you gave vit.D to patients because the ones taking 'cod' had higher levels of vitamin D3 in their blood and were doing better (hypothesis!), you could get poor results because you are not giving together Vit.A and a bit of omega-3 fatty acids, the original recipe.
Do we know what the level of 25(OH)D is among patients newly diagnosed? For that matter do we know how or if 25(OH)D3 influences lymphocyte populations. The reason I ask is I have been reading quite a bit lately about using the ABSOLUTE LYMPHOCYTE COUNT during induction chemotherapy as a way to identify patients at "risk" of relapse and survival.
Do we know what the level of 25(OH)D is among patients newly diagnosed? For that matter do we know how or if 25(OH)D3 influences lymphocyte populations. The reason I ask is I have been reading quite a bit lately about using the ABSOLUTE LYMPHOCYTE COUNT during induction chemotherapy as a way to identify patients at "risk" of relapse and survival.CONCLUSIONS: "ALC is a simple, statistically powerful measurement for patients with de novo AML and ALL. The results, when combined with previous studies, demonstrate that ALC is a powerful new prognostic factor for a range of malignancies. These findings suggest a need for further exploration of postchemotherapy immune status and immune-modulating cancer therapies. Cancer 2008. © 2007 American Cancer Society." Cancer Volume 112, Issue 2 , Pages 407 - 415
...A massive vitamin D 'tsunami' is coming closer,spinning out of the scientific literature circuit:will flu vaccination campaigns be the first casualties?Epidemic influenza and vitamin D.Cannell JJ, Vieth R, Umhau JC, Holick MF, Grant WB, Madronich S, Garland CF, Giovannucci E. Atascadero State Hospital, 10333 El Camino Real, Atascadero, CA 93422, USA.In 1981, R. Edgar Hope-Simpson proposed that a 'seasonal stimulus' intimately associated with solar radiation explained the remarkable seasonality of epidemic influenza. Solar radiation triggers robust seasonal vitamin D production in the skin; vitamin D deficiency is common in the winter, and activated vitamin D, 1,25(OH)2D, a steroid hormone, has profound effects on human immunity. 1,25(OH)2D acts as an immune system modulator, preventing excessive expression of inflammatory cytokines and increasing the 'oxidative burst' potential of macrophages. Perhaps most importantly, it dramatically stimulates the expression of potent anti-microbial peptides, which exist in neutrophils, monocytes, natural killer cells, and in epithelial cells lining the respiratory tract where they play a major role in protecting the lung from infection. Volunteers inoculated with live attenuated influenza virus are more likely to develop fever and serological evidence of an immune response in the winter. Vitamin D deficiency predisposes children to respiratory infections. Ultraviolet radiation (either from artificial sources or from sunlight) reduces the incidence of viral respiratory infections, as does cod liver oil (which contains vitamin D). An interventional study showed that vitamin D reduces the incidence of respiratory infections in children. We conclude that vitamin D, or lack of it, may be Hope-Simpson's 'seasonal stimulus'.Epidemiol Infect. 2006 Dec;134(6):1129-40. Epub 2006 Sep 7.Note: ... Vitamin D deficiency predisposes children to respiratory infections .from: Rickets Today - Children Still Need Milk and SunshineNicholas Bishop,M.D. University of Sheffield...Rickets may have severe consequences. It is strongly associated with pneumonia in young children in developing countries. In a case–control study at the Ethio-Swedish Children's Hospital in Addis Ababa,3 Muhe and colleagues demonstrated an incidence of rickets among children with pneumonia that was 13 times as high as that among control children, after adjustment for family size, birth order, crowding, and months of exclusive breast-feeding. The relative risk of death for the children with rickets as compared with the children without rickets was 1.7. Furthermore, bony deformity of the pelvis in women leads to obstructed labor and increased perinatal morbidity and mortality....Children in developed countries need calcium, too. There is clear evidence from prospective studies of dietary supplementation that increased calcium intake during childhood results in increased calcium retention and increased bone mass.8 Young adults with a history of greater milk consumption have a higher total-body bone mass than those with lower intake after the influence of body size is taken into account.9 Calcium, vitamin D, and phosphate are essential nutrients for the growing skeleton. Wherever children live, they should follow Grandma's advice: "Drink up your milk, and go play outside." N.Engl.J.Med. 1999 341(: 602-604.http://www.goworldtravel.com/ex/aspx/articleGuid.c009fecc-3cc7-4134-b9d5-d203d0111eb3/xe/article.htm
1. Genetic changes seen in patients with leukemia have been demonstrated clonal changes among WBC's in the so called "Guthrie cards" now routinely collected at birth to "screen" for treatable genetic diseases. This work has been widely reported and thought to support the Knudson "two-hit" theory of cancer development - meaning it takes two separate but cooperating events to cause cancer.my point and questions this... could these "clonal changes" and Vitamin D deficiency cooperate to PUSH and individual toward leukemia. The obvious argument against this is that not all patients have demonstrated "clonal" changes. But could this be a CLUE?
2. If you consider the simple CBC differential as a person ages... there is a predicted "switch" that occurs in children between the ages of 2-4 where the number or relative percentage of lymphocytes goes from a dominate position of say 50-60% of the total WBC's to a much lower 30-40%. This may not be related and I certainly don't know the answer why this happens - but it has always "bothered" me as there must be a logical if not scientific reason... the deeper question here is ...Most children develop leukemia during this same time period...the thought I have had for years is what is the connection....if any.
3. If we accept that Vitamin D deficiency is only the tip of the problem, I think it has been stated that overall vitamin D plays many roles but roughly divided you can say one is BONE development and two roughly lumped together "CELL SIGNALING" mechanisms. This brings me to my question about the possible association of Vitamin D and OSTEOSARCOMA.Is it possible that during the adolescent growth spurt the relative amount of vitamin D available for "CELL SIGNALING" events is decreased - thereby creating a "deficiency state" that promotes bone tumor formation?
We know from the 'Shanghai report' that daily doses of vitamins A and D (actually cod liver oil!) -taken for at least one year- could be able to reduce leukemia incidence to half or 1/3.It's not much, but we (parents) should give it a chance and offer this protection to our sick children, to avert relapse risk.
"Physicians and other health care practitioners would be able to treat patients with doses of vitamin D that possess greater therapeutic value than those currently being used while avoiding the risk of adverse effects by administering vitamin D together with vitamins A and K."Med Hypotheses. 2007;68(5):1026-34.
I am a little busy right now I will properly reply as I have more time to compile my thoughts. Sorry - not lack of interest just time.
...Treatment of vitamin D deficiency with 1,25(OH2)D (calcitriol) or analogues of 1,25(OH2)D (paricalcitol, doxercalciferol)are inappropriate, ineffective, dangerous and contraindicated.JJ Cannell et al. 2008 excellent and concise review available full-text online! http://www.vitamindcouncil.org/PDFs/diagnosis-vitdd.pdfQuoteDiagnosis and treatment of vitamin D deficiency. Cannell JJ, Hollis BW, Zasloff M, Heaney RP.Atascadero State Hospital, 10333 El Camino Real, Atascadero, California 93422, USA. jcannell@ash.dmh.ca.govThe recent discovery--in a randomised, controlled trial--that daily ingestion of 1100 IU of colecalciferol (vitamin D) over a 4-year period dramatically reduced the incidence of non-skin cancers makes it difficult to overstate the potential medical, social and economic implications of treating vitamin D deficiency. Not only are such deficiencies common, probably the rule, vitamin D deficiency stands implicated in a host of diseases other than cancer. The metabolic product of vitamin D is a potent, pleiotropic, repair and maintenance, secosteroid hormone that targets > 200 human genes in a wide variety of tissues, meaning it has as many mechanisms of action as genes it targets. A common misconception is that government agencies designed present intake recommendations to prevent or treat vitamin D deficiency. They did not. Instead, they are guidelines to prevent particular metabolic bone diseases. Official recommendations were never designed and are not effective in preventing or treating vitamin D deficiency and in no way limit the freedom of the physician--or responsibility--to do so. At this time, assessing serum 25-hydroxy-vitamin D is the only way to make the diagnosis and to assure that treatment is adequate and safe. The authors believe that treatment should be sufficient to maintain levels found in humans living naturally in a sun-rich environment, that is, > 40 ng/ml, year around. Three treatment modalities exist: sunlight, artificial ultraviolet B radiation or supplementation. All treatment modalities have their potential risks and benefits. Benefits of all treatment modalities outweigh potential risks and greatly outweigh the risk of no treatment. As a prolonged 'vitamin D winter', centred on the winter solstice, occurs at many temperate latitudes, < or = 5000 IU (125 microg) of vitamin D/day may be required in obese, aged and/or dark-skinned patients to maintain adequate levels during the winter, a dose that makes many physicians uncomfortable.Expert Opin Pharmacother. 2008 Jan;9(1):107-18.
Diagnosis and treatment of vitamin D deficiency. Cannell JJ, Hollis BW, Zasloff M, Heaney RP.Atascadero State Hospital, 10333 El Camino Real, Atascadero, California 93422, USA. jcannell@ash.dmh.ca.govThe recent discovery--in a randomised, controlled trial--that daily ingestion of 1100 IU of colecalciferol (vitamin D) over a 4-year period dramatically reduced the incidence of non-skin cancers makes it difficult to overstate the potential medical, social and economic implications of treating vitamin D deficiency. Not only are such deficiencies common, probably the rule, vitamin D deficiency stands implicated in a host of diseases other than cancer. The metabolic product of vitamin D is a potent, pleiotropic, repair and maintenance, secosteroid hormone that targets > 200 human genes in a wide variety of tissues, meaning it has as many mechanisms of action as genes it targets. A common misconception is that government agencies designed present intake recommendations to prevent or treat vitamin D deficiency. They did not. Instead, they are guidelines to prevent particular metabolic bone diseases. Official recommendations were never designed and are not effective in preventing or treating vitamin D deficiency and in no way limit the freedom of the physician--or responsibility--to do so. At this time, assessing serum 25-hydroxy-vitamin D is the only way to make the diagnosis and to assure that treatment is adequate and safe. The authors believe that treatment should be sufficient to maintain levels found in humans living naturally in a sun-rich environment, that is, > 40 ng/ml, year around. Three treatment modalities exist: sunlight, artificial ultraviolet B radiation or supplementation. All treatment modalities have their potential risks and benefits. Benefits of all treatment modalities outweigh potential risks and greatly outweigh the risk of no treatment. As a prolonged 'vitamin D winter', centred on the winter solstice, occurs at many temperate latitudes, < or = 5000 IU (125 microg) of vitamin D/day may be required in obese, aged and/or dark-skinned patients to maintain adequate levels during the winter, a dose that makes many physicians uncomfortable.Expert Opin Pharmacother. 2008 Jan;9(1):107-18.
"Keep away from the sun to avoid cancer""multivitamin pills shorten your life"
...To the authors' knowledge, plaintiffs' attorneys are not yet involved in the vitamin D debate. After the findings of Lappe et al. (1), it may only be a matter of time until lawsuits against physicians begin to appear, claiming that physicians dispensed sun-avoidance advice, but negligently failed to diagnose and treat the consequent vitamin D deficiency, leading to fatal cancers. Unless the future literature fails to support the present, such medical malpractice suits may become commonplace.Finally, physicians and policy-makers should understand that much of the future of vitamin D is out of their hands. Inexpensive high-dose supplements are now widely available to the American public over-the-counter and to the world via the Internet. Sunlight remains free. A Google search for 'vitamin D' reveals several million hits. After the Canadian Cancer Society recently recommended 1000 IU/day for all Canadian adults in the wintertime, vitamin D disappeared off the shelves, causing a shortage during the summer.The pleiotropic actions and unique pharmacology of vitamin D mean educated patients, on their own, can entirely control their own tissue levels of this steroid, through either UVB exposure or over-the-counter supplementation. Given the attitudes that some in mainstream medicine have about any substance with the word 'vitamin' in it (105), the public and not the medical profession may be the first to enter the vitamin D era.
...Getting close…In late 1999 a team of Finnish pediatricians investigate bone turn over in children suffering from cancer (40% leukemias) at completion of therapy. They find abnormal data related to calcium and bone metabolism that explain the high incidence of osteoporosis and pathological fractures observed in these patients. Together with calcium, vitamin D is found significantly lower (P<0.0001). These alterations are referred to bone invasion by cancer initially, but most of all to chemotherapy damage later. These Authors suggest to consider a controlled clinical trial to evaluate the possibility of vitamin D and calcium supplementation.Click down here to see the abstract:http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&dopt=AbstractPlus&list_uids=10531569&query_hl=2&itool=pubmed_docsumSuprisingly, in 1999, writing from the very same country (Finland) the bright hematologist T.T.Timonen gets published in Ann.Hematol. "A hypothesis concerning deficiency of sunlight, cold temperature, and influenza epidemics associated with the onset of acute lymphoblastic leukemia in northern Finland." In the end of the summary: "is hypothesized that sunlight deprivation in the arctic winter can lead to a deficiency of the 1, 25(OH)2D3 vitamin, which might stimulate leukemic cell proliferation and block cell differentiation through dysregulation of growth factors in the bone marrow stromal cells, causing one mutation and an overt ALL in progenitor cells damaged during the current or the previous winter by influenza virus, the other mutation."http://www.yukonhelmut.de/Winter/Artic1.jpg"A hypothesis concerning deficiency of sunlight, cold temperature, and influenza epidemics associated with the onset of acute lymphoblastic leukemia in northern Finland." by T.T. Timonen, 1999.Click down here to see the abstract:http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&dopt=AbstractPlus&list_uids=10525828&query_hl=6&itool=pubmed_docsum...but all this is just supporting Mel Greaves’s hypothesis: “the final hit may be infectious”.Dr. Timo Timonen actually introduces the concept that vitamin D3 deficiency itself might cause leukemia in some patients.http://www.corecharacter.com/uploads/einstein3-thumb.jpg"The whole of science is nothing more than a refinement of everyday thinking."Albert Einstein
Quote...The pleiotropic actions and unique pharmacology of vitamin D mean educated patients, on their own, can entirely control their own tissue levels of this steroid, through either UVB exposure or over-the-counter supplementation. Given the attitudes that some in mainstream medicine have about any substance with the word 'vitamin' in it, the public and not the medical profession may be the first to enter the vitamin D era.John Jacob Cannellhttp://www.vitamindcouncil.org/PDFs/diagnosis-vitdd.pdf..."educated patients" doesn't necessarily mean that they had read up on vitamin D.In some cases personal experience helped to solve this problem from the very start.Many years ago, at the camping ground near the seaside where I went with my family, I noticed four elderly men around a table, playing cards.One of the group was 'reeeally' tanned, almost black, much darker than the others. Another friend passed by and started chatting.After having said to the overtanned fellow: "You surely took a lot of sun this summer!",he got this quick reply, probably the same given every day to others, over and over:"Well, when I don't do this, I get aches and pains in the winter"That wise old man had found out something important for his health all by himself.He actually tested it "on his own skin"... year after year, then he drew his conclusions.No 1988 Shanghai report, no 1999 Timo Timonen's hypothesis, and much before the "resurrection of vitamin D" (2007). http://www.equilibriarte.org/upload/forum/070805145950-238.jpg
...The pleiotropic actions and unique pharmacology of vitamin D mean educated patients, on their own, can entirely control their own tissue levels of this steroid, through either UVB exposure or over-the-counter supplementation. Given the attitudes that some in mainstream medicine have about any substance with the word 'vitamin' in it, the public and not the medical profession may be the first to enter the vitamin D era.John Jacob Cannellhttp://www.vitamindcouncil.org/PDFs/diagnosis-vitdd.pdf
Quote from: neilep on 21/05/2008 16:01:37Quote from: iko on 21/05/2008 10:01:40Easy question Neilepus amicissimus,the answer MIGHT be right here:http://journals.cambridge.org/action/displayAbstract?fromPage=online&aid=529704EnjoyikodAWESOME !!!..Vitamin D Rules !!...If only I knew a good source of Vitamin D !! [] []Hi Neilepus rapidofastissimus thread-makerus!Cod liver oil is no good for 'boosting' your vitamin D: plenty of vitamin A and omega-3 fatty acids plus 'some' vitamin D.It probably works in the long run as far as vitamin D3 is concerned (approx. 400 I.U./day).So dear old 'cod' is still used daily in northern Europe, during months with the 'R': from September to April.Short 'flashes' (30min.) of sushine between 10a.m. to 2p.m. at a proper latitude (no clouds please) really boost your skin production of vitamin D3 (>20,000 I.U.)Sorry you cannot read the complete paper previously cited by ikus.You would be impressed by a 1918 study about flu reported in the article.Shortly, in 1918, trying to find out how influenza viruses managed to infect people and to verify relative incubation times (2-3 days), proper experiments were set up using human volunteers. Forget the details [xx(]...but secretions from infected patients were carefully collected, mixed up and flushed through the nostrils of brave volunteers. []Surprisingly enough, nothing happened afterwards, so the experiment was considered a 'fiasco'.Only now, 90 years later, a crystal-clear explanation is ready for this.Anti-infective properties of vitamin D were proved only 4-5 years ago, when the cathelicidin pathway was described.Those volunteers were healthy men from the Navy.Probably well-tanned all year round, perfectly healthy, they had been selected for not having had a flu in the previous months, to avoid an 'immunization' bias.Maybe a good level of vitamin D helped them to block the influenza viruses quickly.For the same reason, somewhere in 2005, most (maybe all) vitamin D supplemented patients in Dr J.Cannell department, Atascadero CA, went through a big influenza epidemic perfectly healthy.John Cannell was the 'prepared mind', times were changing, so the vitamindcouncil.com crusade started.One hypothesis out of many is that flu viruses do circulate all year round in humans, but give troubles in some people only in the cold season, i.e. when vitamin D levels are low.So much for the anti-flu vaccination campaigns.http://www.youtube.com/watch?v=enB6BuOjXY8P.S.The reason why Chris is not commenting on these issues is simple: he is a virology expert and knows much better than others the other side of the coin.Everybody is waiting for final scientific proofs about vitamin D and flu, but most of all about vitamin D benefits in other dreadful diseases.Wonderful hypotheses need extended and accurate studies to become Science. It takes so much time.I'm sure Chris will never have to decide from trembling hypotheses whether to give 'cod' to one of his kids or NOT.It happened to me, after years of serious searches and rigorous evidence-based training.That's life.ikodHypothesis--ultraviolet-B irradiance and vitamin D reduce the risk of viral infections and thus their sequelae, including autoimmune diseases and some cancers.Grant WB.Sunlight, Nutrition, and Health Research Center, San Francisco, CA, USA. wgrant@infionline.netMany viral infections reach clinical significance in winter, when it is cold, relative humidity is lowest and vitamin D production from solar ultraviolet-B irradiation is at its nadir. Several autoimmune diseases, such as multiple sclerosis, type 1 diabetes mellitus and asthma, are linked to viral infections. Vitamin D, through induction of cathelicidin, which effectively combats both bacterial and viral infections, may reduce the risk of several autoimmune diseases and cancers by reducing the development of viral infections. Some types of cancer are also linked to viral infections. The cancers with seemingly important risk from viral infections important in winter, based on correlations with increasing latitude in the United States, an index of wintertime solar ultraviolet-B dose and vitamin D, are bladder, prostate, testicular and thyroid cancer, Hodgkin's and non-Hodgkin's lymphoma, and, perhaps, gastric cancer. The evidence examined includes the role of viruses in the etiology of these diseases, the geographic and seasonal variation of these diseases, and the time of life when vitamin D is effective in reducing the risk of disease. In general, the evidence supports the hypothesis. However, further work is required to evaluate this hypothesis.Photochem Photobiol. 2008 Mar-Apr;84(2):356-65. Epub 2008 Jan 7.
Quote from: iko on 21/05/2008 10:01:40Easy question Neilepus amicissimus,the answer MIGHT be right here:http://journals.cambridge.org/action/displayAbstract?fromPage=online&aid=529704EnjoyikodAWESOME !!!..Vitamin D Rules !!...If only I knew a good source of Vitamin D !! [] []
Easy question Neilepus amicissimus,the answer MIGHT be right here:http://journals.cambridge.org/action/displayAbstract?fromPage=online&aid=529704Enjoyikod
M. A. Helou, G. Massey, G. Francis, K. Godder, J. Laver Abstract: Background: Survivors of childhood cancer are at increased risk for osteoporosis. Contributing factors include direct effects of chemotherapy and radiation therapy on bone, secondary hormone deficiencies, and chronic illness. However, vitamin D insufficiency could be a major risk factor during and after cancer therapy. Vitamin D insufficiency is common in healthy school aged children (median 25-hydroxy vitamin D [25(OH)D] = 28 ng/mL, 55% <30 ng/mL, 5% < 10 ng/mL.) Based on this data, we hypothesize that vitamin D insufficiency would be common among children with cancer. If vitamin D insufficiency is prevalent, correction may contribute to better bone health and immune responses in children with cancer. Methods: We determined the serum levels of 25(OH)D, PTH, calcium, and phosphorus for 40 children with leukemia or lymphoma currently on therapy (group 1), 34 children with leukemia or lymphoma off therapy (group 2), 16 children with solid tumors currently on therapy (group 3), and 10 children with solid tumors off therapy (group 4.) Prevalence of 25(OH)D insufficiency ( <32 ng/mL) and severe deficiency (<10 ng/mL) was compared by Chi square test to the healthy reference population (established by Weng, et al.)Results: For the majority of patients, calcium and phosphorus levels were within normal limits. Conclusions: Vitamin D insufficiency was very common in all groups, especially in children with solid tumors on therapy (Group 3.) 25(OH)D levels did improve off therapy, but for Group 2, still remained significantly less than normal reference population (p=0.0001.) The data suggests that vitamin D status should be determined for all children at diagnosis of malignancy with a strong recommendation to consider vitamin D supplementation during treatment and follow up. J Clin Oncol 26: 2008 (May 20 suppl; abstr 10023)
Unfortunately, if vitamin D is needed mainly, and too much vitamin A is either toxic or counteracting "D" wonderful effects (J.Cannell et al. Nov.2008), we would need a special cod liver oil formula:a moderate amount of vitamin A, plenty of D-vitamin and lots of omega-3! This probably WAS the original cod liver oil, before they started removing D-vitamin, erroneously thinking that it was too close to toxic amounts.Two thousands I.U. per day of vitamin D3 was considered almost toxic for humans.What a shame: we seem to have destroyed the original formula.
From January 2008 VitaminD Newsletter:Quote...All of the epidemiological and animal studies in the literature suggest cancer patients will prolong their lives if they take vitamin D. I can't find any studies that indicate otherwise. However, none of the suggestive studies are randomized controlled interventional trials; they are all epidemiological or animal studies, or, in the case of Vieth's, an open human study. However, if you have cancer, or your child does, do you want to wait the decades it will take for the American Cancer Society to fund randomized controlled trials using the proper dose of vitamin D? Chances are you, or your child, will not be around to see the results. John Cannell, MDhttp://content.ll-0.com/vitalchoiceseafood/Cannell.John.140.jpghttp://www.vitamindcouncil.com/http://www.lung.ca/tb/images/full_archive/006_codLiverOil.jpghttp://karmadaze.com/sunrise.jpg
...All of the epidemiological and animal studies in the literature suggest cancer patients will prolong their lives if they take vitamin D. I can't find any studies that indicate otherwise. However, none of the suggestive studies are randomized controlled interventional trials; they are all epidemiological or animal studies, or, in the case of Vieth's, an open human study. However, if you have cancer, or your child does, do you want to wait the decades it will take for the American Cancer Society to fund randomized controlled trials using the proper dose of vitamin D? Chances are you, or your child, will not be around to see the results. John Cannell, MDhttp://content.ll-0.com/vitalchoiceseafood/Cannell.John.140.jpghttp://www.vitamindcouncil.com/