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It's never too late (sometimes)...If you followed this thread so far,you deserve to watch this free video:"The Vitamin D Pandemic and its Health Consequences"Presented by Michael Holick, PhD, MD, Professor of medicine, physiology and biophysicsand director of the General Clinical Research Center at Boston University Medical CenterKeynote address at the opening ceremony of the 34th European Symposium on Calcified Tissues, Copenhagen 5 May, 2007http://www.uvadvantage.org/portals/0/pres/http://www.uvadvantage.org/portals/0/pres/video/video/slides/slide414.jpg
Video of Professor Michael Holick explaining the vitamin D revolution.Michael Holick, a respected Vitamin D researcher recently gave keynote address about Vitamin D. It is a great overview of the entire situation. Watch the video- The Vitamin D Pandemic and its Health Consequences
...It's not a miracle pill. But early indications suggest when it comes to health measures you can control, taking vitamin D may rank up there with quitting smoking.Yet nearly all Canadians are probably deficient in this so-called "sunshine" vitamin, the only vitamin you can't get in large amounts from a good diet.And much of this information has been staring scientists in the face, generally unrecognized, for 40 years or more....http://www.canada.com/vancouversun/news/story.html?id=2afe61fa-d92f-426d-a401-5611c495e418&p=1
If you like videos, here is Dr. John Cannell's interview on CBNnews.comwhich I just found searching in a gold mine (vitamindcouncil):http://www.cbn.com/CBNnews/269033.aspxhttp://www.trackyourplaque.com/library/images/Cannell100.jpg
Hi Carolyn! Thanks for calling...Cod liver oil is not exactly like fish oilNote:fish liver oils contain vit.A+Vit.D3+omega-3 fatty acids.fish oilscontain omega-3 fatty acids mainly.Vit.A&Vit.D3 are fat-soluble and do accumulate in the body: doses higher than reccomended may lead to toxicity.ikoSo when you give fish oil you are giving omega-3 fatty acids mainly.Omega-3 may have amazing effects on the human brain and they are currently being tested in patients with bipolar disorder (a type of psychotic depression): there are positive and negative reports, as usual. They just started few years ago...it is a promising field.There is an interesting book about this by a pharmacology professor, Andrew L. Stoll:http://www.amazon.com/Omega-3-Connection-Groundbreaking-Anti-depression-Program/dp/0684871386In proper doses you may be sure to give good nutrients (they come from the sea plankton!) and no toxicity. Even if they don't work, your kid will be safe and well nourished.I don't know ADHD enough to reccomend anything, but speaking of nutritional supplement and reminding the paper cited at the beginning of this topic, I would suggest to alternate fish oil with CLO.Vitamin D has also positive effects on the brain.Check carefully expiring date and storage reccomended conditions: these oils may go rancid quick.Control of the nervous systemVitamin D3 actions in the nervous system include induction of Vitamin D Receptor content (VDR is expressed in the brain and on several regions of the central and peripheral nervous system), the conductance velocity of motor neurons, and the synthesis of neurotrophic factors, such as nerve growth factors and neurotrophyns, that prevent the loss of injured neurons. Vit.D3 also enhances the expression of glial cell line-derived neurotrophic factor, a potential candidate for treatment of Parkinson's disease.In addition to increased nerve growth factor, combined treatment with Vit.D3 and 17beta-estradiol in rats elicits neuroprotective effects after focal cortical ischemia induced through the photothrombosis model.Vit.D3 influences critical components of orderly brain development. In the embrionic rat brain, the VDR increases steadly from day 15 to day 23, and Vit.D3 induces the expression of nerve growth factor and stimulates neurite outgrowth in embryonic hippocampal explants and primary cultures. Low prenatal Vitamin D in utero leads to increased brain size, brain shape, enlarged ventricles, and reduced expression of nerve growth factor in the neonatal rat.The association of vit.D deficiency and abnormal brain development makes Vitamin D an attractive candidate for tretment of schizophrenia, a disorder resulting from gene-environment interactions that disrupt brain development.Also, transient prenatal vitamin D deficiency in rats induces hyperlocomotion in adulthood with sever motor abnormalities.simplified by me from a nice and thick recent review by Adriana S. Dusso and coll.:Vitamin Dhttp://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&dopt=AbstractPlus&list_uids=15951480&query_hl=1&itool=pubmed_docsumShortly, there is increasing experimental evidence that vitamin D is just good for the brain.No negative effects are reported.Take careikoPost Scriptum:I just found a positive report about ADHD and fish oil. You probably started from this one...let me know. There are so many references to get lost between papers and scientific reports.It's in another Forum! What a fantastic gigantic basket this Google is...http://www.feelgoodforum.com/about1564.html...
Iko,Is cod liver oil the same type of Omega-3 oils used (supposedly) for lowering cholesterol?
..."No one is naive," he said. "Vitamin D optimization won't eliminate cancer by any stretch of the imagination, but if it has no downsides and it cuts cancer incidence, it could be worthwhile. Nobody wants to overlook a clue here. This is what everybody wants - a simple pill that reduces cancer risk."
"Sit down before fact as a little child, be prepared to give up every preconceived notion, follow humbly wherever and whatever abysses nature leads,or you will learn nothing."Thomas Henry Huxley
Quote from: iko on 28/04/2008 21:51:47We know from the 'Shanghai report' that daily doses of vitamins A and D (actually cod liver oil!) -taken for at least one year- could be able to reduce leukemia incidence to half or 1/3.It's not much, but we (parents) should give it a chance and offer this protection to our sick children, to avert relapse risk. Yes, an autoquote.Waiting for cod4ALL and his comments on the 'Mansoura report' and those striking data about vitamin D deficiency at 0-3-12 months after a diagnosis of childhood leukemia (47 cases: further studies are obviously needed...in the next 2-3 decades).for at least one yearWhy such a long lag-phase of cod-therapy is needed before achieving a significant anti-leukemia effect? Could this be bound mainly to a vitamin D action?Well, simple minds can only give simple answers:If proper levels of vitamin D are needed to counteract leukemia onset, a deficient child (and most humans seem to be vitamin D deficient these days!) gets a relatively small amount of the sunshine vitamin from daily doses of cod liver oil. Maybe vitamin A helps, together with omega-3 fatty acids and vitamin E, but daily vitamin D through 'cod' is about 400 I.U.Not much to treat deficiency, so it should take longer to work. Maybe vitamin A helpsThis is a recent medical hypothesis: vitamin A could prevent toxicity of vitamin D and cooperate with vitamin K too...all the fat-soluble vitamins together!Isn't it wonderful? []Vitamin D toxicity redefined: vitamin K and the molecular mechanism. Masterjohn C.Weston A. Price Foundation, 4200 Wisconsin Ave., NW, Washington, DC 20016, United States. ChrisMasterjohn@gmail.comThe dose of vitamin D that some researchers recommend as optimally therapeutic exceeds that officially recognized as safe by a factor of two; it is therefore important to determine the precise mechanism by which excessive doses of vitamin D exert toxicity so that physicians and other health care practitioners may understand how to use optimally therapeutic doses of this vitamin without the risk of adverse effects. Although the toxicity of vitamin D has conventionally been attributed to its induction of hypercalcemia, animal studies show that the toxic endpoints observed in response to hypervitaminosis D such as anorexia, lethargy, growth retardation, bone resorption, soft tissue calcification, and death can be dissociated from the hypercalcemia that usually accompanies them, demanding that an alternative explanation for the mechanism of vitamin D toxicity be developed.The hypothesis presented in this paper proposes the novel understanding that vitamin D exerts toxicity by inducing a deficiency of vitamin K. According to this model, vitamin D increases the expression of proteins whose activation depends on vitamin K-mediated carboxylation; as the demand for carboxylation increases, the pool of vitamin K is depleted. Since vitamin K is essential to the nervous system and plays important roles in protecting against bone loss and calcification of the peripheral soft tissues, its deficiency results in the symptoms associated with hypervitaminosis D. This hypothesis is circumstantially supported by the observation that animals deficient in vitamin K or vitamin K-dependent proteins exhibit remarkable similarities to animals fed toxic doses of vitamin D, and the observation that vitamin D and the vitamin K-inhibitor Warfarin have similar toxicity profiles and exert toxicity synergistically when combined. The hypothesis further proposes that vitamin A protects against the toxicity of vitamin D by decreasing the expression of vitamin K-dependent proteins and thereby exerting a vitamin K-sparing effect. If animal experiments can confirm this hypothesis, the models by which the maximum safe dose is determined would need to be revised. Physicians and other health care practitioners would be able to treat patients with doses of vitamin D that possess greater therapeutic value than those currently being used while avoiding the risk of adverse effects by administering vitamin D together with vitamins A and K.Med Hypotheses. 2007;68(5):1026-34.New recipeQuote"Physicians and other health care practitioners would be able to treat patients with doses of vitamin D that possess greater therapeutic value than those currently being used while avoiding the risk of adverse effects by administering vitamin D together with vitamins A and K."Med Hypotheses. 2007;68(5):1026-34.And here we go with cod liver oil (containing vitamins A and D) plus spinaches, cabbage, cauliflower, and other green leafy vegetables (rich of vitamin K)!We might even give rosemary (carnosic acid) and sesamolin a chance.And never forget orange juice! (but this is another story) []
We know from the 'Shanghai report' that daily doses of vitamins A and D (actually cod liver oil!) -taken for at least one year- could be able to reduce leukemia incidence to half or 1/3.It's not much, but we (parents) should give it a chance and offer this protection to our sick children, to avert relapse risk.
"Physicians and other health care practitioners would be able to treat patients with doses of vitamin D that possess greater therapeutic value than those currently being used while avoiding the risk of adverse effects by administering vitamin D together with vitamins A and K."Med Hypotheses. 2007;68(5):1026-34.
Could by any chance the old remedy, a relic from the past,an inexpensive nutrient containing vitamins A and D,help leukemic patients in the long run, AFTER treatment?According to this recent study, the answer is yes.Differentiation-inducing liposoluble vitamin deficiency may explainfrequent secondary solid tumors after hematopoietic stem cell transplantationMinireview.Gedikoglu G, Altinoz MA.Secondary cancers are among the most threatening long-term health problems of hematopoetic stem cell- transplant (HSCT) patients. There are several lines of evidence indicating the possibility of a prolonged Vitamin A deficiency for solid tumor-type secondary cancers: I- Solid tumors such as oral cavity, head/neck region squamous carcinomas, skin cancers and melanomas, where lowered Vitamin A concentrations and chemo-preventing activity of its derivatives (retinoids) are most explicitly proven, arise much more frequently than others. II- Early monitorings: A significant retinol deficiency in HSCT patients is detectable along with a severity of mucositis and the vulnerability to infection. III- Monitoring of other liposoluble vitamins: Vitamin D, a differentiation-inducing vitamin like Vitamin A, showed a sustained decrease. Another similarity of these two vitamins is that they also depend on intestinal absorption and are decreased due to bowel injury by conditioning agents and chronic graft-versus-host disease. IV- Peroxidative reactions and inflammation can directly exhaust retinol levels despite sufficient intake. Considering the similar inhibitory role of Vitamin D analogs (deltanoids) on squamous carcinomas, skin tumors and melanomas, we propose that animal studies and extended vitamin surveillance studies in HSCT patients may unfold a preventive strategy against long-term complications.Neoplasma. 2008;55(1):1-9.
There are many stories of mothers forcing their children to take cod liver oil.Centuries ago, northern Europeans used cod liver oil to protect them from the cold. It was made from the livers of Gadus morhua and other species of cod. Cod liver oil was said to relieve such complaints as rheumatism, aching joints, and stiff muscles.At the beginning of the 20th century, scientists established that cod liver oil was antirachitic, and it became commonplace for mothers to give it to their children.[1,2]It turns out cod liver oil contains large amounts of vitamins A, D, and omega-3 fatty acids, and the health benefits may go beyond rheumatism and rickets.[3]...